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Epidermal growth factor receptor gene mutations and increased copy numbers predict gefitinib sensitivity in patients with recurrent non-small-cell lung cancer.

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none of the biomarkers predicted differential overall survival between gefitinib and docetaxel

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First-line Indolent (FIT) study presented at the American Society of Hematology (ASH) Annual Meeting demonstrated the continued improvement in progression-free survival (PFS) following ibritumomab consolidation therapy for patients with follicular B-cell non-Hodgkin's lymphoma who achieved a response to first-line therapy over chemotherapy alone.

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Assessment of EGFR mutation status in lung adenocarcinoma by immunohistochemistry using antibodies specific to the two major forms of mutant EGFR.

Based on available data, it is not entirely clear if the order of use of EGFR TKI is significant.

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EZH2 () inhibition has differential effects on the TopoII inhibitor response of nonsmall-cell lung cancers in vitro and in vivo.

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the brain metastasis in this patient was also reduced with the increased gefitinib dosages

Gefitinib patients had fewer serious AEs (22.

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Surgical resection is the treatment of choice for stage II NSCLC, except for those patients who are not surgical candidates because of comorbid conditions or poor pulmonary reserve.

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All but one of these deaths were considered unrelated to treatment; one patient in the gefitinib group died from drug-related hepatic and renal failure.

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This increase may be clinically relevant as adverse experiences are related to dose and exposure; therefore, caution should be used when administering CYP3A4 inhibitors with IRESSA (see and sections).

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Mapatumumab and lexatumumab induce apoptosis in TRAIL-R1 and TRAIL-R2 antibody-resistant NSCLC cell lines when treated in combination with bortezomib.

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Determine the local progression-free survival and disease-specific survival (cancer vs co-morbid disease) of patients treated with this regimen.

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The bulk of prospective clinical data regarding chemotherapy and molecularly targeted therapy for elderly NSCLC patients come from studies in advanced disease.

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Although any risk from air pollution is very small, it nevertheless may be a contributor to those lung cancers not obviously related to smoking.

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Accordingly, the choice of which sample to test should be based primarily on the specimen qualities themselves (tumor content and preservation), rather than whether they are primary or metastatic lesions.

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N1 disease, whereas for patients with stage III, N2 disease there was no clear evidence of an adverse effect.

Until high level evidence demonstrates that routine surveillance with PET or PET-CT scans helps prolong life or promote well-being after treatment for a specific type of cancer, this practice should not be done.

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Eligible patients were administered gefitinib and celecoxib, both given orally, from day 1 until disease progression, unacceptable toxicity, or withdrawal.

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The starting dose of 300 mg per day was selected on the basis of its efficacy in a parallel phase 1 study in patients with CML in the chronic phase.

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The first non-T790M secondary mutation described was D761Y.

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Insights gained from the treatment of patients with metastatic NSCLC with gefitinib and erlotinib are dramatically changing drug development and treatment strategies, as well as clinical outcomes.

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Based on the present results and published data, SRS alone as initial treatment strategy in patients with either single or multiple metastases is a feasible therapeutic option associated with high local control and survival benefits, although the superiority of SRS versus other treatment options in terms of improved survival remains to be demonstrated.

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Phase III study showed benefits of maintenance use of pemetrexed for non-squamous NSCLC.

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D1 reduction caused by EGFR inhibitors was investigated.

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Rebiopsy of lung cancer patients with acquired resistance to EGFR inhibitors and enhanced detection of the T790M mutation using a locked nucleic acid-based assay.

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Surveillance, Epidemiology, and End Results (SEER) data suggest that the percentage of patients aged older than 70 years is closer to 50%.

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At this point, gefitinib was stopped, the patient provided written informed consent, and a transbronchial aspirate and transbronchial biopsy specimen were obtained.

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Hypercalcemia: Some lung cancers produce substances that remove calcium from bone and release it into the bloodstream, causing a condition called hypercalcemia.

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An alternative serial blood and tumor microdialysis study design, as we have used previously (, ), was ruled out due to the poor dialyzability of gefitinib and the need to obtain a sufficient tumor mass for both drug and PD measurements.

Gefitinib is active in patients with brain metastases from non-small cell lung cancer and response is related to skin toxicity.

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Erlotinib and gefitinib are very important drugs for patients with NSCLC harboring sensitive mutations of EGFR.

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Biochemical analyses of transfected cells and growth inhibition studies with lung cancer cell lines demonstrate that the T790M mutation confers resistance to EGFR mutants usually sensitive to either gefitinib or erlotinib.

Pooled safety analysis of EGFR-TKI treatment for EGFR mutation-positive non-small cell lung cancer.

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As an inhibitor of PDGFR, imatinib mesylate appears to have utility in the treatment of a variety of dermatological diseases.

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Randomized trials of conventional radiation therapy versus SBRT (), and hypofractionated radiation therapy versus SBRT () are ongoing to determine the optimal radiation therapy regimen, but stereotactic body radiation therapy has been widely adopted for patients with medically inoperable stage I NSCLC.

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