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none of the biomarkers predicted differential overall survival between gefitinib and docetaxel
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EZH2 () inhibition has differential effects on the TopoII inhibitor response of nonsmall-cell lung cancers in vitro and in vivo.
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the brain metastasis in this patient was also reduced with the increased gefitinib dosages
Gefitinib patients had fewer serious AEs (22.
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Surgical resection is the treatment of choice for stage II NSCLC, except for those patients who are not surgical candidates because of comorbid conditions or poor pulmonary reserve.
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All but one of these deaths were considered unrelated to treatment; one patient in the gefitinib group died from drug-related hepatic and renal failure.
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This increase may be clinically relevant as adverse experiences are related to dose and exposure; therefore, caution should be used when administering CYP3A4 inhibitors with IRESSA (see and sections).
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Mapatumumab and lexatumumab induce apoptosis in TRAIL-R1 and TRAIL-R2 antibody-resistant NSCLC cell lines when treated in combination with bortezomib.
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The bulk of prospective clinical data regarding chemotherapy and molecularly targeted therapy for elderly NSCLC patients come from studies in advanced disease.
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Although any risk from air pollution is very small, it nevertheless may be a contributor to those lung cancers not obviously related to smoking.
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Accordingly, the choice of which sample to test should be based primarily on the specimen qualities themselves (tumor content and preservation), rather than whether they are primary or metastatic lesions.
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N1 disease, whereas for patients with stage III, N2 disease there was no clear evidence of an adverse effect.
Until high level evidence demonstrates that routine surveillance with PET or PET-CT scans helps prolong life or promote well-being after treatment for a specific type of cancer, this practice should not be done.
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Eligible patients were administered gefitinib and celecoxib, both given orally, from day 1 until disease progression, unacceptable toxicity, or withdrawal.
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Insights gained from the treatment of patients with metastatic NSCLC with gefitinib and erlotinib are dramatically changing drug development and treatment strategies, as well as clinical outcomes.
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Based on the present results and published data, SRS alone as initial treatment strategy in patients with either single or multiple metastases is a feasible therapeutic option associated with high local control and survival benefits, although the superiority of SRS versus other treatment options in terms of improved survival remains to be demonstrated.
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An alternative serial blood and tumor microdialysis study design, as we have used previously (, ), was ruled out due to the poor dialyzability of gefitinib and the need to obtain a sufficient tumor mass for both drug and PD measurements.
Gefitinib is active in patients with brain metastases from non-small cell lung cancer and response is related to skin toxicity.
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Erlotinib and gefitinib are very important drugs for patients with NSCLC harboring sensitive mutations of EGFR.
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Comparative genomic sequence analysis and isolation of human and mouse alternative EGFR transcripts encoding truncated receptor isoforms.
Biochemical analyses of transfected cells and growth inhibition studies with lung cancer cell lines demonstrate that the T790M mutation confers resistance to EGFR mutants usually sensitive to either gefitinib or erlotinib.
Pooled safety analysis of EGFR-TKI treatment for EGFR mutation-positive non-small cell lung cancer.
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